From 663657735479a92d80eeb73248d27068b1bca98c Mon Sep 17 00:00:00 2001 From: Benito Whitford Date: Wed, 24 Sep 2025 11:24:03 +0800 Subject: [PATCH] Add Glycogen Storage Disease (GSD) --- Glycogen-Storage-Disease-%28GSD%29.md | 1 + 1 file changed, 1 insertion(+) create mode 100644 Glycogen-Storage-Disease-%28GSD%29.md diff --git a/Glycogen-Storage-Disease-%28GSD%29.md b/Glycogen-Storage-Disease-%28GSD%29.md new file mode 100644 index 0000000..8d52d0c --- /dev/null +++ b/Glycogen-Storage-Disease-%28GSD%29.md @@ -0,0 +1 @@ +
Such a scheme serves as a template for the lactate shuttling present in the mammalian CNS. An virtually contemporaneous report prolonged this scheme to use to the mammalian mind, but with notable differences and additions. It was to prove some of the contentious papers in the sector of brain vitality metabolism and the controversy raging round the topic persists. The paper described experiments carried out using cultures of mouse cerebral neurones and astrocytes, and demonstrated a couple of key options, which have been stoichiometrically sturdy (Pellerin and Magistretti, 1994). The background to the research was the realization that if metabolic signaling were to happen between neurones and astrocytes in the mammalian CNS, there must exist a signaling mechanism whereby the neurones sign to astrocytes their need for supply of metabolic substrate. The signaling mechanism will need to have the following characteristics: (1) it have to be related to the frequency of activity of the neurones i.e., the discharge of the compound have to be associated to the exercise of the neurones, (2) it have to be sensed by the astrocyte i.e., there should be mechanism(s) whereby the astrocyte senses the molecule, and (3) there must exist a system or process whereby the concentration of the molecule is decreased upon cessation of activity i.e., there should be an effective buffering system that reduces the concentration of the molecule after exercise.

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